Manganese Supplements and Dystonia

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Mutations in SLC39A14 disrupt manganese homeostasis and cause childhood-onset parkinsonism–dystonia

Although manganese is an essential trace metal, little is known about its transport and homeostatic regulation. Here we have identified a cohort of patients with a novel autosomal recessive manganese transporter defect caused by mutations in SLC39A14. Excessive accumulation of manganese in these patients results in rapidly progressive childhood-onset parkinsonism-dystonia with distinctive brain...

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BACKGROUND The first gene causing early-onset generalized dystonia with brain manganese accumulation has recently been identified. Mutations in the SLC30A10 gene, encoding a manganese transporter, cause a syndrome of hepatic cirrhosis, dystonia, polycythemia, and hypermanganesemia. METHODS We present 10-year longitudinal clinical features, MRI data, and treatment response to chelation therapy...

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ژورنال

عنوان ژورنال: Pediatric Neurology Briefs

سال: 1995

ISSN: 2166-6482,1043-3155

DOI: 10.15844/pedneurbriefs-9-1-6